SOLUTION\u00a0PROPECIA\u00a0HAIR LOSS <\/strong><\/p>\n Why\u00a0Hair Loss\u00a0Sudden\u00a0or Slow? That\u00a0many questions\u00a0that\u00a0arise\u00a0every person who\u00a0is found in\u00a0this state with premature\u00a0hair loss.What is\u00a0normality?<\/p>\n Why\u00a0we\u00a0are born\u00a0with this or that\u00a0capital\u00a0hair?<\/p>\n The\u00a0hair seems\u00a0little influenced by\u00a0the external environment and\u00a0the transmission of their\u00a0main characteristics\u00a0is largely\u00a0genetic.<\/p>\n However, as\u00a0the nails, they\u00a0consist mainly\u00a0of protein (keratin)\u00a0and their health\u00a0isinfluenced by\u00a0food hygiene\u00a0of the person.<\/p>\n Drug\u00a0for baldness<\/strong><\/p>\n Propecia\u00a0contains the active ingredient,\u00a0finasteride, known as\u00a0Alpha\u00a0reduct ase inhibitor\u00a0type II\u00a05.<\/span><\/p>\n Propecia\u00a0blocks the action\u00a0of\u00a0alpha\u00a0reductase type\u00a0II\u00a05\u00a0and thus\u00a0the formation of DHT.\u00a0The decrease in\u00a0the amount of\u00a0DHT\u00a0in the blood and\u00a0the scalp\u00a0can lead to stopping\u00a0the process of\u00a0hair loss.<\/p>\n Propecia\u00a0increases\u00a0hair growth\u00a0and prevents\u00a0hair loss\u00a0due to\u00a0male pattern baldness.\u00a0Once\u00a0started treatment\u00a0with Propecia, it may take\u00a03 to 6 monthsbefore you\u00a0notice a\u00a0real decrease\u00a0in the loss of\u00a0hair\u00a0or increased\u00a0density.Continued use of\u00a0this drug\u00a0is recommended for\u00a0maximum results.<\/p>\n France.euroClinix.net <\/p>\n<\/div>\n MEN:<\/p>\n Women:<\/strong><\/p>\n Androgenetic Alopecia<\/strong><\/span><\/p>\n What\u00a0is it?<\/strong><\/p>\n Hair loss due to two factors: a hormonal factor, and a hereditary factor.<\/p>\n Genetic susceptibility\u00a0of the scalp\u00a0androgen\u00a0(male hormone\u00a0whose main subject is, in this case, testosterone).\u00a0So this is\u00a0a predisposition\u00a0in the field.<\/p>\n It\u00a0may be more\u00a0commonly known as\u00a0seborrheic alopecia\u00a0or\u00a0baldness\u00a0\u00ab\u00a0classic\u00a0\u00bbbecause it\u00a0affects 90% of\u00a0men with\u00a0alopecia<\/p>\n She moved\u00a0slowly and\u00a0diffusely.<\/p>\n It is\u00a0directly related to\u00a0male hormones\u00a0in the blood, but\u00a0mainly affects\u00a0the scalp.These are the\u00a0hair follicles\u00a0of the\u00a0frontal areas,\u00a0parietal\u00a0(temples) and the\u00a0top of the skull\u00a0that are most\u00a0sensitive to these\u00a0hormonal disturbances.<\/p>\n Androgenetic alopecia\u00a0in women\u00a0appeared\u00a0there\u00a0twenty years.\u00a0Since\u00a0it\u00a0affects a\u00a0growing number<\/p>\n In women\u00a0androgenetic alopecia\u00a0is a\u00a0refinement\u00a0of the hair.\u00a0It is less spectacular\u00a0than in men, focusing\u00a0on the top\u00a0of the skull\u00a0or on the\u00a0line.<\/p>\n The\u00a0human body\u00a0produces\u00a0androgens\u00a0as well as that\u00a0of women\u00a0(20\u00a0times lessthan in men).\u00a0All of the\u00a0androgen\u00a0is secreted by\u00a0the gonads (testes and\u00a0ovaries, 40%)\u00a0and adrenal glands\u00a0(60%).\u00a0These hormones\u00a0have many\u00a0functions but\u00a0for both sexes\u00a0and there is\u00a0a\u00a0genetic susceptibility,\u00a0they accelerate\u00a0and shorten\u00a0the life cycle\u00a0of hair follicles.<\/p>\n In the case of\u00a0a\u00a0genetic\u00a0hypersensitivity, the\u00a0hair becomes\u00a0finer\u00a0and shorter.Then,\u00a0exhausted\u00a0bulbs\u00a0shrink, producing\u00a0more than a\u00a0fine down, that’s whatleads\u00a0to baldness<\/p>\n Family history is\u00a0a\u00a0critical factor in determining\u00a0androgenetic alopecia.<\/p>\n Explanations\u00a0of a\u00a0physiological point of view<\/strong><\/span><\/p>\n <\/p>\n On the scalp, testosterone is converted into dihydrotestosterone (DHT) under the influence of the enzyme 5\u03b1-reductase. DHT stimulates the sebaceous glands which increase sebum production. This effect is particularly apparent in adolescence. Stagnant sebum in the hair follicle will gradually clog. Therefore,the scalp thicken, preventing blood vessels to feed the bulb.<\/p>\n Another\u00a0negative effect\u00a0of DHT\u00a0is\u00a0known\u00a0to shorten\u00a0the growth phase\u00a0of hairand\u00a0its life cycle, which makes\u00a0the hair\u00a0thinner and more\u00a0fragile.<\/p>\n Is the transmission\u00a0of genes\u00a0from the mother\u00a0alopecia\u00a0or\u00a0father,\u00a0or both, both\u00a0in boys\u00a0than in girls,\u00a0that causes this\u00a0genetic predisposition.<\/p>\n These genes\u00a0stimulate the activity\u00a0of\u00a05\u00a0alpha-reductase,\u00a0which in turn\u00a0leads to amuch greater\u00a0secretion\u00a0of DHT. If there is no\u00a0genetic predisposition, the two\u00a0substances\u00a0combine\u00a0harmless anddo not interfere\u00a0in any way the\u00a0normal\u00a0physiological development\u00a0of the hair.<\/p>\n Androgenetic Alopecia\u00a0in Women<\/span><\/strong><\/p>\n <\/p>\n Family history is a key factor in the diagnosis of androgenetic alopecia.<\/p>\n But more than 20% of women with no history in their family and yet have clinical signs of this alop\u00e9cie.Arround 30% of women suffer from androgenetic alopecia.<\/p>\n This predisposition is associated with hypersensitivity to androgens and hyperactivity of the enzyme 5\u03b1-reductase.\u00a0As humans in this case, it will be imperative to act locally (at the scalp) on androgen production and 5\u03b1-reductase.<\/p>\n It is very important to act at the first sign of abnormal fall, because once used up any capital hair back will never be possible.\u00a0Care for external application can improve the appearance of hair, however it is not enough to guarantee the integrity of hair follicles.\u00a0An internal processing-time, which if necessary could be renewed at certain times of life, is absolutely essential.\u00a0However, it is very important to know the type of fall which the person is facing in order to choose the treatment best suited fall.<\/p>\n Androgenetic alopecia is very easy to diagnose in women: the hair is thinning only on the top of the head and sometimes on the temples, while the hair of the neck\u00a0and temples is dense and well stocked.\u00a0This alopecia begins with a widening of the median line.\u00a0The hair becomes thinner and sometimes it goes hand in hand with seborrhoea.\u00a0It is irreversible if we do not address the problem.<\/p>\n<\/div>\n Androgenetic alopecia\u00a0in men<\/strong><\/span><\/p>\n <\/p>\n This is the most common hair loss in men.<\/p>\n It affects both young men from the age of 20-22 years (20%) than men in their forties\u00a050% of men from the age of 50 suffer from androgenetic alopecia.\u00a0This alopecia is a hereditary predisposition that is to say, it is usually transmitted from father to son.\u00a0This does not mean that all men suffering from androgenetic alopecia have a child predisposed to this type of alopecia.\u00a0This is related to hyperactivity of the enzyme 5\u03b1-reductase, it is essential to regulate, but it is impossible to completely inhibit because it is inevitably linked to the production of testosterone.<\/p>\n It recognizes the hair loss androgenetic its location.\u00a0Bare areas are the upper front (gulfs bilateral) or the top of the skull, the fall in the area usually starts with a tonsure, while the neck and temples remain dense and well furnished.\u00a0This decline stems from the overactive alphareductase 5.<\/p>\n This enzyme will convert locally and excessive testosterone to dihydrotestosterone (DHT).\u00a0Rate too high will cause DHT shortens the growth phase of the hair.<\/p>\n The length of each hair cycle will significantly decrease from a few years to months.\u00a0The hair cycles, normally scheduled to last a lifetime, will therefore run out much faster.\u00a0Excess DHT usually causes increased activity of the sebaceous glands surrounding the hair bulbs.\u00a0This excess seborrhea will gradually clog the pores of the bulb that slowly suffocate.\u00a0Meanwhile, the scalp is not well vascularized, or fed.\u00a0All these phenomena will lead to atrophy of the bulb, resulting in a first thinning hair, and then a gradual and total loss of hair on areas of more extensive scalp.<\/p>\n
\nHow\u00a0about it?
\nIs there\u00a0a\u00a0SOLUTION\u00a0a natural or\u00a0an anti\u00a0Hair loss?<\/p>\n![\"L'image](\"http:\/\/img.bleskovky.sk\/20532.jpg\/bruce-willis.jpg\")
<\/p>\nWe lose our hair every day,and it’s normal.Lose fifty hairs a day is quite normal.But then,if the fall is more important, it is important to respond, because there is a beginning to a specific cause.<\/strong><\/h4>\n
Propecia<\/span><\/h1>\n
How does\u00a0Propecia?<\/h3>\n
Propecia blocks the action of a compound in the body called type II 5 alphareductase. This enzyme is responsible for the conversion of testosterone to dihydrotestosterone (DHT). DHT is a hormone that can damage the hair follicles, causing hair loss in men who are sensitive to this hormone.<\/h4>\n
\nEuropean website\u00a0selling\u00a0the drug\u00a0for\u00a0baldness\u00a0follows:\u00a0Propecia<\/a><\/strong><\/span><\/p>\n<\/h1>\n
![\"Acheter](\"https:\/\/www.hexpressaffiliates.com\/accounts\/default1\/banners\/468x60-hairloss-fr.gif\" "\\"Acheter")
<\/p>\nExplanation of\u00a0the life\u00a0of a hair<\/h1>\n
Their number: between\u00a090,000\u00a0and 150,000<\/span><\/h4>\n
Their origin,\u00a0their\u00a0roots\u00a0are\u00a0from the\u00a0follicles or\u00a0hair follicles\u00a0that\u00a0are between 15 and\u00a025 cycles\u00a0of life<\/span><\/h3>\n
Each\u00a0bulb\u00a0is attached to\u00a0the sebaceous gland\u00a0(which develops\u00a0especially afterpuberty)<\/span><\/h3>\n
The transmission\u00a0characteristics of a\u00a0hair is\u00a0mostly\u00a0genetic<\/span><\/h3>\n
The diameter\u00a0of a human hair\u00a0varies between\u00a00.05\u00a0mm\u00a0and 0.1\u00a0mm<\/span><\/h3>\n
Each\u00a0life cycle\u00a0of a human hair\u00a0is well defined\u00a0there are\u00a0three\u00a0important phases of\u00a0the life\u00a0of our hair:<\/span><\/h3>\n
Most of our\u00a0hair is\u00a0in a growth phase\u00a0(that\u00a0is, they\u00a0are between\u00a02 and 5\u00a0years).This is the\u00a0longest\u00a0phase\u00a0of the life\u00a0of the hair,\u00a0in which\u00a0each hair\u00a0can grow\u00a0from 0.75 to\u00a01.5 cm\u00a0per month.<\/span><\/h3>\n
At this\u00a0growth phase\u00a0followed by a\u00a0preparation phase of\u00a0three weeks:\u00a0the hair\u00a0is getting ready for\u00a0the resting phase<\/span><\/h3>\n
A resting phase\u00a0of\u00a03 months\u00a0which leads to\u00a0hair loss.<\/span><\/h3>\n
Then\u00a0the cycle begins again:\u00a0a new hair\u00a0regrowth and\u00a0replaces\u00a0one that\u00a0fell.
\n<\/span><\/h3>\n<\/div>\n<\/div>\nALL the RISKS<\/h3>\n
<\/p>\n
About 65% of men\u00a0experiencing\u00a0hair loss\u00a0problem<\/strong><\/h2>\n
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\nThese\u00a0hair loss\u00a0can start\u00a0as young as\u00a018 years, they reach\u00a0one in three men\u00a0at thirty,\u00a0and nearly one\u00a0in two men\u00a0around\u00a0fifty.<\/p>\n
\n<\/span><\/p>\n<\/li>\n<\/ul>\n<\/div>\n<\/h1>\n
About 70% of women are faced with a problem of hair loss in a period of their lives, but they are generally favored a view to hormonal menopause or pre-menopause.
\nSome women more sensitive on a genetic point of view, can suffer from diffuse alopecia: hair, in this case, become very fine.<\/h3>\n
\n<\/span><\/h1>\n<\/div>\n<\/div>\n![\"\"](\"http:\/\/tbn0.google.com\/images?q=tbn:4AbvwpJo9e5pQM:http:\/\/img221.imageshack.us\/img221\/6003\/pelade9ms.png\")
<\/a><\/div>\n
\nWe must therefore\u00a0understand\u00a0that in this case, it is not\u00a0the amount of testosterone\u00a0secreted\u00a0is in question, but\u00a0how\u00a0\u00ab\u00a0abnormal\u00a0\u00bb\u00a0it is used\u00a0and converted\u00a0by the\u00a0genetically programmed\u00a0hair follicles.\u00a0In other words, it\u00a0is not\u00a0a hormonal disorder, but\u00a0an abnormal\u00a0receptor\u00a0bulbs.<\/p>\n